Abstract：
The research aims to evaluate the seasonal differences in the distribution, source, and risks of water-contaminated PAHs. The PAHs were extracted by the liquid-liquid method and analyzed with GC-MS, and a total of eight PAHs were detected. There was a percentage increase in the average concentration of the PAHs from the wet to the dry season in the range of 20 (Anthracene)-350 (Pyrene)%. Total PAHs (∑PAHs) range from 0.31 to 1.23 mg/l in the wet period and from 0.42 to 1.96 mg/l in the dry period. The distribution of the average PAHs in mg/l showed that Fluoranthene ≤ Pyrene < Acenaphthene < Fluorene < Phenanthrene < Acenaphthylene < Anthracene < Naphthalene in wet period and while Fluoranthene < Acenaphthene < Pyrene < Fluorene < Phenanthrene < Acenaphthylene < Anthracene < Naphthalene in the dry period. The children were exposed to non-carcinogenic risk through non-dietary ingestion due to the accumulative effect (HI) of the PAHs in the dry period. Furthermore, the naphthalene was responsible for ecological and carcinogenic risk in the wet period, while the fluorene, phenanthrene, and anthracene were responsible for ecological and carcinogenic risk in the dry period. However, while adults and children are both susceptible to carcinogenic risk through the oral channel during the dry period, only children are susceptible to non-carcinogenic risk through this pathway. The multivariate statistical analysis revealed the influence of physicochemical parameters on the detected PAHs and also showed the PAHs\' sources to be mainly combustion, pyrolysis, and vehicular emission.
摘要：
该研究旨在评估分布的季节性差异，来源,和水污染的PAHs的风险。采用液-液法提取PAHs,用GC-MS分析,共检测到8种PAHs。从雨季到旱季，PAHs的平均浓度在20（蒽）-350（芘）％的范围内增加了百分比。湿期的总PAHs（∑PAHs）范围为0.31至1.23mg/l，干期为0.42至1.96mg/l。以mg/l为单位的平均PAHs分布表明，在潮湿时期，氟蒽≤Pyrene<芴<芴<菲<乙炔<蒽<萘，而在干燥时期，氟蒽<乙炔<芘<芴<菲<蒽<萘。由于在干旱期PAHs的累积作用（HI），儿童通过非饮食摄入而暴露于非致癌风险。此外，萘在潮湿时期是生态和致癌风险的原因，而芴，菲,和蒽是干旱期生态和致癌风险的原因。然而,虽然成人和儿童在干旱期都容易通过口腔通道致癌风险，独生子女通过这一途径易受非致癌风险的影响.多元统计分析揭示了物理化学参数对检测到的PAHs的影响，也表明PAHs的来源主要是燃烧，热解,和车辆排放。
