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  • 文章类型: Case Reports
    Pre-eclampsia complicates approximately 5-8% of all pregnancies and may have adverse long-term effects on both mother and child. Chronic atraumatic subdural haematoma as a complication of severe pre-eclampsia, in the absence of clotting factor abnormalities, is a very rare condition. We present the case of a 30-year-old Moroccan woman who had a pregnancy 10 years previously, with an uneventful delivery. She presented with pre-eclampsia complicating a 29-week-old pregnancy. A few days preceding maternity unit admission the patient complained of headaches and malaise. Her blood pressure at admission was 150/120mmHg and subsequently was treated with doses of methyldopa and magnesium sulphate. Her condition worsened with a loss of consciousness 24 hours later and was transferred to the neurosurgical unit. A brain computerized tomography (CT) scan revealed a left-sided subdural haematoma and the patient underwent surgery, with a good postoperative outcome. This article highlights the occurrence of neurological complications due to pre-eclampsia/eclampsia that require particular neurosurgical attention, its treatment and prognosis. We also review the literature regarding this pathology.
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  • 文章类型: Case Reports
    在先兆子痫中,电解质紊乱如低钙血症和/或低钠血症的发生是不常见的事件,这可能是情况严重的迹象,对母亲和她的父母有潜在的不利后果。子痫前期的低钠血症是严重程度的指标,并且需要了解病因机制以开始适当的治疗。事实上,经常考虑的液体限制很少是孕妇的治疗选择。低钙血症是一种并发症,当采用高剂量的静脉内硫酸镁治疗时,必须进行监测。在这种情况下,必须寻求低钙血症,排除其他病因如维生素D缺乏,甲状旁腺功能减退或肾脏和肾外钙的损失。替代疗法,静脉内或口服根据情况,在严重或症状性低钙血症的情况下应考虑。
    The occurrence of electrolyte disorders as hypocalcemia and/or hyponatremia is an uncommon event in preeclampsia, which can be the sign of serious situation, with potentially unfavourable consequences for the mother and her fœtus. Hyponatremia in the setting of preeclampsia is an indicator of severity, and requires the understanding of the etiologic mechanisms to initiate an appropriate treatment. Indeed the often-considered fluid restriction is rarely a treatment option for pregnant women. Hypocalcemia is a complication that must be monitored when a treatment with high doses of intravenous magnesium sulphate is introduced. In this context, hypocalcemia must be sought, with the exclusion of other etiologies as vitamin D deficiency, hypoparathyroidism or renal and extrarenal loss of calcium. A replacement therapy, intravenous or oral according to circumstances, should be considered in case of severe or symptomatic hypocalcemia.
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