背景:本案例研究的目的是确定Osgood-Schlatter病(OSD)的双侧病因因素以及支持12岁精英女性奥林匹克Karateka治疗过程有效性的因素。
方法:本案例研究涉及OSD女性空手道,她在4岁时开始了运动训练。
方法:整形外科医生的主观触诊和超声检查的客观体检结果,墙壁滑动试验,磁共振成像,收集身高和体重测量值。
方法:运动员膝关节的治疗干预持续了20个月(5个阶段)。物理治疗,运动疗法,并给予药物治疗,体育活动逐渐引入。
结果:身高发育轨迹一致,体重不稳定。OSD在第二次生长突增后被诊断出来,在随后的身高和体重增加以及运动训练的体积和强度增加期间,报告了显着的进展。从髌骨韧带到胫骨突的距离变化的速率和动力学是不规则的,右膝占优势,变化率最高(-3.3mm),变化率消退的两倍(-2.5mm)。分析距离从未超过基线值(5.5mm),左膝就是这种情况。从治疗的第二个月开始就可以重返体育比赛,其中运动疗法和静态拉伸是最有效的。在身高和体重增加稳定时,记录了the骨韧带与胫骨骨的相对正确距离。OSD后未观察到病理变化,并观察到完全恢复。
结论:在本研究中讨论的情况下,生长突增,这项运动的特殊性,早期专业化,包括高容量和高强度训练应被视为导致OSD及其进展的因素。运动疗法管理和静态拉伸对于OSD的治疗至关重要。由于早期的治疗干预,有可能快速恢复体育比赛,这也可能导致胫骨结节没有病理变化和OSD没有复发。
BACKGROUND: The purpose of this
case study was to identify factors of bilateral etiopathogenesis of Osgood-Schlatter disease (OSD) and those supporting the effectiveness of the therapeutic process in a 12-year-old elite female Olympic karateka.
METHODS: The present
case study concerns OSD female karateka who started her sport-specific training at the age of 4 years.
METHODS: The results of subjective palpation by the orthopedic surgeon and objective medical examination using ultrasonography, wall slide test, magnetic resonance imaging, and body height and weight measurements were collected.
METHODS: The therapeutic intervention for the athlete\'s knee joints lasted 20 months (5 stages). Physical therapy, kinesiotherapy, and pharmacological treatment were administered, and physical activity was gradually introduced.
RESULTS: The developmental trajectory was uniform for body height and labile for body weight. OSD was diagnosed after the second growth spurt, and significant progression was reported during the subsequent height and weight gains and increased volume and intensity of sports training. The rate and dynamics of changes in the distance from the patellar ligament to the tibial apophysis were irregular, with dominance in the right knee with the highest rate of change (-3.3 mm) and twice the regression of the rate of change (-2.5 mm). The analyzed distance never exceeded the baseline value (5.5 mm), which was the
case in the left knee. Return to sports competition was possible from the second month of therapy, in which kinesiotherapy and static stretching were the most effective. A relatively correct distance of the patellar ligament from the tibial apophysis was recorded at the time of stabilization of the body height and weight gain. No pathological changes were observed following OSD, and full recovery was observed.
CONCLUSIONS: In the
case discussed in this study, growth spurt, the specificity of the sport practiced, and early specialization including high-volume and high-intensity training should be considered as factors causing OSD and its progression. Kinesiotherapeutic management and static stretching are crucial for the treatment of OSD. Quick return to sports competition was possible due to early therapeutic intervention, which could also lead to the absence of pathological changes in the tibial tubercle and the absence of recurrence of OSD.