Abstract：
We investigated whether a single heart rate clamped cycling session under systemic hypoxia affects the recovery of physical and psycho-physiological responses from residual fatigue compared to normoxia. On separate occasions, twelve trained males performed a 3-d acute training camp scenario. On days 1 and 3, participants cycled for 60 min at a constant heart rate (80% of ventilatory threshold). On day 2, fatigue was induced through a simulated team game circuit (STGC), followed by a 60-min intervention of either: (1) heart rate clamped cycling in normoxia; (2) heart rate clamped cycling in hypoxia (simulated altitude ~ 3500 m); or (3) no cycling. Countermovement jump height and leg stiffness were assessed before and after every session. Perceptual fatigue was evaluated daily. Compared to baseline, jump height decreased at all timepoints following the STGC (all p < 0.05). Leg stiffness and cycling power output only decreased immediately following the STGC, with a 48% further decrease in cycling power output in hypoxia compared to normoxia (p < 0.05). Perceived fatigue, decreased sleep quality, and increased muscle soreness responses occurred on day 3 (p < 0.05). A single heart rate-clamped cycling session in hypoxia reduced mechanical output without affecting recovery of physical performance and perceptual measures from residual fatigue induced through team sport activity.
摘要：
我们调查了与正常缺氧相比，在全身缺氧下单个心率钳制的循环过程是否会影响残余疲劳的身体和心理生理反应的恢复。在不同的场合，12名受过训练的男性进行了3-d急性训练营场景。在第1天和第3天，参与者以恒定的心率（通气阈值的80％）循环60分钟。在第2天，通过模拟团队游戏电路（STGC）引起疲劳，随后进行60分钟的干预：（1）常氧条件下的心率钳制循环；（2）低氧条件下的心率钳制循环（模拟海拔〜3500m）；或（3）无循环。在每次训练前后评估反运动跳跃高度和腿部刚度。每天评估感知疲劳。与基线相比，在STGC之后，跳跃高度在所有时间点都下降（所有p<0.05）。腿部刚度和循环功率输出仅在STGC之后立即降低，与常氧相比，低氧条件下的循环功率输出进一步降低了48％（p<0.05）。感知疲劳，睡眠质量下降，增加的肌肉酸痛反应发生在第3天(p<0.05)。在缺氧条件下进行一次心率钳制的循环训练会减少机械输出，而不会影响身体表现的恢复以及团队运动引起的残余疲劳的感知措施。
