我们介绍了一例涉及一名30多岁的男性患者的病例,该患者在过去5年中因低钾血症反复发作而入院。他的病史显示高血压,注意缺陷多动障碍(ADHD),自闭症,和偏执狂。他在服用西酞普兰,雷米普利,氨氯地平,还有普拉克索.测试表明醛固酮/肾素比率和血浆肾上腺素水平正常。回顾他的饮食史,据指出,他每天消耗3至3.5升可乐味饮料。在可乐味饮料的摄入量和钾替代量显着减少后,钾水平达到了正常水平。随后的门诊随访显示,即使患者停止服用钾替代片剂,钾水平仍保持正常。鉴于与碳酸饮料相关的低钾血症的罕见,这种关联的潜在机制尚不清楚.在这个案例报告中,我们试图为所涉及的机制提供一个可能的解释。
We present a case involving a male patient in his 30s who was admitted to hospital due to recurrent episodes of hypokalaemia over the past 5 years. His medical history revealed hypertension, attention deficit hyperactivity disorder (ADHD), autism, and paranoia. He was taking citalopram, ramipril, amlodipine, and pramipexole. Tests indicated normal levels of aldosterone/renin ratio and plasma metanephrines. On reviewing his dietary history, it was noted that he consumed 3 to 3.5 L of cola-flavoured drinks on a daily basis. Normal potassium levels were achieved after a significant reduction in cola-flavoured drinks intake and potassium replacement. Subsequent outpatient clinic follow-up revealed that normal potassium levels were maintained even after the patient ceased taking potassium replacement tablets. Given the rarity of hypokalaemia associated with fizzy drinks, the underlying mechanism for this association remains unclear. In this case report, we attempt to provide a possible explanation for the involved mechanisms.