未经证实:胎盘是怀孕期间的短暂器官,胎儿与子宫壁相连.怀孕经常并发妊娠期糖尿病,这可能会导致胎盘的形态变化(重量,直径,和子叶数量);因此,它可能会影响胎儿和母亲。
UNASSIGNED:这项研究的目的是确定妊娠糖尿病孕妇与未妊娠糖尿病孕妇之间胎盘子叶数量的差异,然后将其与组之间的重量和直径相关联。
UNASSIGNED:一项比较研究(妊娠糖尿病A组和非妊娠糖尿病B组)包括在接受知情同意后足月(37-40周)分娩的单胎婴儿的母亲。排除患有孕前糖尿病和其他慢性疾病的妇女以及宫内胎儿死亡的妇女。分娩后胎盘被准确地准备和详细检查。胎盘重量,直径,用SPSS21版记录和分析子叶数。在子叶计数方面测量两组之间的相关性,胎盘直径,和重量。
UNASSIGNED:该研究包括385名参与者(A组128名,B组257名)。子叶的胎盘数量,体重,A组的直径高于B组,差异有统计学意义(P=0.000,P=0.021,P=0.000)。A组,胎盘重量之间存在显着相关性,直径,子叶数(r=0.23,P=0.011)。子叶计数受糖尿病控制的影响显着(P=0.021)。
未经证实:妊娠糖尿病增加胎盘子叶数量,体重,和直径。
UNASSIGNED: Placenta is a transient organ during pregnancy, connects the fetus to the uterine wall. Pregnancy is frequently complicated by gestational diabetes, which might cause morphological changes in the placenta (weight, diameter, and
cotyledons number); consequently, it may affect both fetus and mother.
UNASSIGNED: The aim of this study was to determine the difference in placental
cotyledons number between pregnant with gestational diabetes versus without gestational diabetes, then correlate it with the weight and diameter between groups.
UNASSIGNED: A comparative study (gestational diabetes Group A and nongestational diabetes Group B) included mothers with a singleton baby delivered at term (37-40 weeks) after acceptance of the informed consent. Women with pregestational diabetes and other chronic diseases and those with intrauterine fetal death were excluded. Postdelivery placentae were accurately prepared and examined in detail. The placental weight, diameter, and
cotyledons number were recorded and analyzed by SPSS version 21. The correlation was measured between the two groups in terms of
cotyledons count, placental diameter, and weight.
UNASSIGNED: The study included 385 participants (128 Group A and 257 Group B). Placental number of
cotyledons, weight, and diameter in Group A were higher than in Group B, and the difference was significant (P = 0.000, P = 0.021, and P = 0.000, respectively). In Group A, there was a significant correlation between the placental weight, diameter, and number of its cotyledons (r = 0.23, P = 0.011). Cotyledon count was significantly affected by diabetic control (P = 0.021).
UNASSIGNED: Gestational diabetes increases placental cotyledons number, weight, and diameter.