未经评估:慢性疼痛和抑郁和/或焦虑症状都会对日常生活产生负面影响,包括干扰和缺乏生活控制。然而,人们对疼痛和心理困扰如何影响这些影响知之甚少。
未经评估:第一个目的是评估疼痛强度,心理困扰,社会支持与干扰和缺乏生活控制相互作用。第二个目的是调查这些关系的强度是否受到抑郁和/或焦虑的存在或不存在的调节。
未经评估:患者报告的结果测量(PROM),可在瑞典疼痛康复质量注册中心(SQRP)中获得,已检索到慢性疼痛患者(N=40,184)。具有构造/潜在变量疼痛强度的理论模型,心理困扰,干扰,缺乏生活控制,利用偏最小二乘结构方程模型(PLS-SEM)提出并分析了社会支持。从SQRP的PROM中鉴定出这些构建体的指标。总队列的两个模型,在疼痛强度和心理困扰之间的因果关系方面有所不同,被调查了。还分析了焦虑和/或抑郁的调节作用。
UNASSIGNED:发现疼痛强度-心理困扰关系的相关性和解释力相对较低(R2=0.16)。与缺乏生活控制相比,疼痛强度对干扰的影响更大。发现相反的心理困扰-即,心理困扰对功能的负面影响似乎高于对干扰的负面影响。疼痛强度和心理困扰之间因果关系的基本假设决定了强烈的疼痛强度和心理困扰如何影响干扰和缺乏生活控制。社会支持与干扰和缺乏生活控制显示出非常相似的绝对显着相关性。干扰和缺乏生活控制显示出相对较弱的关联。心理困扰水平是所研究的几种路径的调节因素。
UNASSIGNED:疼痛强度和心理困扰之间的低相关性的临床治疗结果可能是临床治疗中的一种可能不会降低另一种的效果。疼痛强度和心理困扰对干扰和缺乏生活控制的相对重要性取决于有关疼痛强度-心理困扰关系的基本假设。干扰和缺乏生活控制表现出相对弱的关联,强调需要单独进行临床评估。社会支持影响了所调查的两个影响结构。队列显示异质性,因此是否存在明确的焦虑和/或抑郁迹象是所研究的几种关联(路径)的调节因素。结果对于慢性疼痛患者的评估和治疗设计都很重要。
UNASSIGNED: Both chronic pain and depressive and/or anxiety symptoms are associated with negative impacts on daily living, including
interference and lack of life control. However, little is known about how pain and psychological distress affect these impacts.
UNASSIGNED: The first aim was to assess how pain intensity, psychological distress, and social support interact with interference and lack of life control. A second aim was to investigate whether the strength of these relationships is moderated by the presence or absence of depression and/or anxiety.
UNASSIGNED: Patient-Reported Outcome Measures (PROMs), which are available in the Swedish Quality Registry for Pain Rehabilitation (SQRP), were retrieved for patients with chronic pain (N = 40,184). A theoretical model with the constructs/latent variables pain intensity, psychological distress, interference, lack of life control, and social support was proposed and analyzed using Partial Least Squares Structural Equation Modelling (PLS-SEM). Indicators for these constructs were identified from the PROMs of the SQRP. Two models of the total cohort, which differed with respect to the causal relationship between pain intensity and psychological distress, were investigated. The moderating effects of anxiety and/or depression were also analyzed.
UNASSIGNED: Relatively low correlation and explanatory power (R 2 = 0.16) were found for the pain intensity-psychological distress relationship. Pain intensity had a stronger effect on
interference than on lack of life control. The reverse was found for psychological distress - i.e., psychological distress seemed to have a higher negative influence on function than on interference. The underlying assumption of the causal relationship between pain intensity and psychological distress determined how strong pain intensity and psychological distress influenced interference and lack of life control. Social support showed very similar absolute significant correlations with
interference and lack of life control.
Interference and lack of life control showed relatively weak associations. The psychological distress level was a moderating factor for several of the paths investigated.
UNASSIGNED: A clinical treatment consequence of the low correlation between pain intensity and psychological distress may be that clinically treating one may not reduce the effect of the other. The relative importance of pain intensity and psychological distress on
interference and lack of life control depends on the underlying assumption concerning the pain intensity-psychological distress relationship. Interference and lack of life control showed relatively weak associations, underscoring the need to clinically assess them separately. Social support influenced both impact constructs investigated. The cohort display heterogeneity and thus presence of definite signs of anxiety and/or depression or not was a moderating factor for several of the associations (paths) investigated. The results are important both for the assessments and the design of treatments for patients with chronic pain.