增加植物特别是作物对各种寒冷胁迫的脆弱性会降低植物生长,发展,产量生产,和植物分布。冷应激诱导生理,形态学,生物化学,表型,和植物的分子变化。转录因子(TF)是介导基因表达的重要调控因子之一。TF是通过信号转导途径激活的,与顺式作用元件一起调节冷响应基因的转录,这些基因有助于提高植物的耐寒性。这里,AP2/ERFTF家族是与其他TF家族一起最重要的冷应激相关TF家族之一,比如WRKY,bHLH,bZIP,MYB,NAC,和C2H2相互关联,以增强耐冷胁迫能力。在过去的十年里,已经发现解决转录因子(TFs)在提高植物耐寒性中的作用的重大进展,比如组学分析。此外,许多研究已经确定和表征了TFs之间或TFs与其他因素(内源性和外源性)之间的冷应激机制的复杂性,包括植物激素,丁香酚,和光。角色,函数,这些TFs之间或TFs与其他增强耐寒性的因素之间的关系仍有待澄清。这里,本研究分析了AP2/ERFTF的作用以及AP2/ERF与MYB之间的联系,WRKY,bZIP,bHLH,C2H2或NAC对冷应力的耐受性。
Increasing the vulnerability of plants especially crops to a wide range of cold stress reduces plant growth, development, yield production, and plant distribution. Cold stress induces physiological, morphological, biochemical, phenotypic, and molecular changes in plants. Transcription factor (TF) is one of the most important regulators that mediate gene expression. TF is activated by the signal transduction pathway, together with cis-acting element modulate the transcription of cold-responsive genes which contribute to increasing cold tolerance in plants. Here, AP2/ERF TF family is one of the most important cold stress-related TF families that along with other TF families, such as WRKY, bHLH, bZIP, MYB, NAC, and C2H2 interrelate to enhance cold stress tolerance. Over the past decade, significant progress has been found to solve the role of transcription factors (TFs) in improving cold tolerance in plants, such as omics analysis. Furthermore, numerous studies have identified and characterized the complexity of cold stress mechanisms among TFs or between TFs and other factors (endogenous and exogenous) including phytohormones, eugenol, and light. The role, function, and relationship among these TFs or between TFs and other factors to enhance cold tolerance still need to be clarified. Here, the current study analysed the role of AP2/ERF TF and the linkages among AP2/ERF with MYB, WRKY, bZIP, bHLH, C2H2, or NAC against cold stress tolerance.