目的:目的是描述声学,听觉感知,喉内收肌张力障碍(AdLD)患者在伦巴第效应(LE)下的主观声音变化。
方法:声音努力的主观感知(OMNI声音努力量表OMNI-VES),最大语音时间(MPT),并在10例AdLD患者和10例典型语音患者的静止状态和LE下评估了发声障碍的感知严重程度(GRBAS量表)。要求扬声器产生持续的元音/a/并大声朗读语音平衡的文本。使用PRAAT软件,分析了以下声学参数:平均音调(Hz),最小和最大强度(dB),局部无声帧的分数,语音中断的次数,语音中断程度(%),倒谱峰突出度平滑(CPPS)(dB)。
结果:在LE下,AdLD组显示GRBAS和主观努力的G和S参数均降低,平均MPT显著增加;在对照组中没有显著变化。在LE下的两组中,持续元音/a/的音高和强度与LE一致显着增加。在AdLD组中,OMNI-VES得分的平均增益和语音分析的每个参数的平均增益均显着大于对照组。
结论:在LE下获得的听觉反馈剥夺改善了主观,知觉-听觉,和AdLD患者的声学参数。这些发现鼓励进一步的研究,为听觉系统在AdLD发病机理中的作用提供新的知识,并开发新的治疗策略。
方法:4喉镜,2024.
OBJECTIVE: The aim was to describe the acoustic, auditory-perceptive, and subjective voice changes under the Lombard effect (LE) in adductor laryngeal dystonia (AdLD) patients.
METHODS: Subjective perception of vocal effort (OMNI Vocal Effort Scale OMNI-VES), Maximum Phonation Time (MPT), and the perceptual severity of dysphonia (GRBAS scale) were assessed in condition of stillness and under LE in 10 AdLD patients and in 10 patients with typical voice. Speakers were asked to produce the sustained vowel /a/ and to read a phonetically balanced text aloud. Using the PRAAT software, the following acoustic parameters were analyzed: Mean Pitch (Hz), Minimum and Maximum Intensity (dB), the Fraction of Locally Unvoiced Frames, the Number of Voice Breaks, the Degree of Voice Breaks (%), the Cepstral Peak Prominence-Smoothed (CPPS) (dB).
RESULTS: Under LE, the AdLD group showed a decrease of both G and S parameters of GRBAS and subjective effort, mean MPT increased significantly; in the controls there were no significant changes. In both groups under LE, pitch and intensity of the sustained vowel /a/ significantly increased consistently with LE. In the AdLD group the mean gain of OMNI-VES score and the mean gain of each parameter of the speech analysis were significantly greater than the controls\' ones.
CONCLUSIONS: Auditory feedback deprivation obtained under LE improves subjective, perceptual-auditory, and acoustics parameters of AdLD patients. These findings encourage further research to provide new knowledge into the role of the auditory system in the pathogenesis of AdLD and to develop new therapeutic strategies.
METHODS: 4 Laryngoscope, 134:3754-3760, 2024.