PDF(Pubmed)
Abstract：
UNASSIGNED: Moderate regular physical activity is indicated to avoid atrial fibrillation (AF), whereas athletes should be counseled that long-lasting vigorous sports engagement may cause AF, according to the 2016 European Society of Cardiology (ESC) recommendations for AF treatment. Exercise and AF are complex.
UNASSIGNED: To evaluate the relationship between Endurance training and AF, in addition to the starting point/trigger by which Endurance Training causes impairment of cardiac function and AF, considering the time and intensity of Endurance training.
UNASSIGNED: We synthesized evidence from articles published in the PubMed, EMBASE, and SciELO databases using their respective Boolean operators. A total of 112 original articles related to AF and endurance athletes published up to the year 2023 were reviewed.
UNASSIGNED: Our study verified multiples aspects of the genesis of AF in athletes, such as cardiac adaptations to exercise, disturbances in cardiac injury biomarkers, sex differences in cardiac adaptations and their role in AF risk, and the relationship between body composition (height, weight, and physical fitness) and AF pathogenesis.
UNASSIGNED: Variations in cardiac structure (increased atrial thickness and size in addition to myocardial fibrosis) and significant increases in vagal tone (sinus bradycardia and imbalances in sympathetic and parasympathetic activation) shorten the refractory period shortening in athletes, induce the onset of re-entrance mechanisms, and serve as ectopic triggers that can lead to AF.
UNASSIGNED: To evaluate the relationship between Endurance training and AF, in addition to the starting point/trigger by which Endurance Training causes impairment of cardiac function and AF, considering the time and intensity of Endurance training.
UNASSIGNED: We synthesized evidence from articles published in the PubMed, EMBASE, and SciELO databases using their respective Boolean operators. A total of 112 original articles related to AF and endurance athletes published up to the year 2023 were reviewed.
UNASSIGNED: Our study verified multiples aspects of the genesis of AF in athletes, such as cardiac adaptations to exercise, disturbances in cardiac injury biomarkers, sex differences in cardiac adaptations and their role in AF risk, and the relationship between body composition (height, weight, and physical fitness) and AF pathogenesis.
UNASSIGNED: Variations in cardiac structure (increased atrial thickness and size in addition to myocardial fibrosis) and significant increases in vagal tone (sinus bradycardia and imbalances in sympathetic and parasympathetic activation) shorten the refractory period shortening in athletes, induce the onset of re-entrance mechanisms, and serve as ectopic triggers that can lead to AF.
摘要：
■应进行适度的有规律的体力活动以避免房颤（AF），而运动员应该被告知，长时间的剧烈运动可能会导致AF，根据2016年欧洲心脏病学会（ESC）对AF治疗的建议。运动和AF是复杂的。
■为了评估耐力训练与AF之间的关系，除了耐力训练导致心脏功能和AF受损的起点/触发因素之外,考虑耐力训练的时间和强度。
■我们从PubMed上发表的文章中综合了证据，EMBASE,和SciELO数据库使用各自的布尔运算符。截至2023年，共发表了112篇与AF和耐力运动员有关的原创文章。
■我们的研究验证了运动员房颤起源的多个方面，比如心脏对运动的适应，心脏损伤生物标志物的紊乱，心脏适应的性别差异及其在房颤风险中的作用，和身体成分(身高，体重,和身体素质）和AF发病机制。
心脏结构的变化（除心肌纤维化外，心房厚度和大小增加）和迷走神经张力的显着增加（窦性心动过缓和交感神经和副交感神经激活的不平衡）缩短了运动员的不应期缩短，诱导重新进入机制的开始，并充当可能导致AF的ectopic触发器。
■为了评估耐力训练与AF之间的关系，除了耐力训练导致心脏功能和AF受损的起点/触发因素之外,考虑耐力训练的时间和强度。
■我们从PubMed上发表的文章中综合了证据，EMBASE,和SciELO数据库使用各自的布尔运算符。截至2023年，共发表了112篇与AF和耐力运动员有关的原创文章。
■我们的研究验证了运动员房颤起源的多个方面，比如心脏对运动的适应，心脏损伤生物标志物的紊乱，心脏适应的性别差异及其在房颤风险中的作用，和身体成分(身高，体重,和身体素质）和AF发病机制。
心脏结构的变化（除心肌纤维化外，心房厚度和大小增加）和迷走神经张力的显着增加（窦性心动过缓和交感神经和副交感神经激活的不平衡）缩短了运动员的不应期缩短，诱导重新进入机制的开始，并充当可能导致AF的ectopic触发器。
