PDF(Pubmed)
Abstract：
BACKGROUND: Heavy metals are one of the most important environmental pollutants in marine coastal ecosystems. Cadmium is a heavy metal that enters to marine environments via industrial wastes and oil production activities.
OBJECTIVE: This study were done to determine the toxicity of cadmium to Litopenaeus vannamei and to evaluate the histological changes in gill tissues after exposure to sublethal concentrations of cadmium at different salinities.
METHODS: For this reason, toxicity test was done to determine the lethal concentration (LC50) of cadmium for whiteleg shrimp. According to the calculated LC50 amount, sublethal doses of cadmium were used to determine its histological effects in different salinity during 2 weeks exposing period.
RESULTS: LC50 of cadmium for 96 h for whiteleg shrimp was 6.56 mg/L. Histological alterations in the gill were observed in L. vannamei after 14 days exposure to different concentrations of cadmium and salinity. Histopathological index was increased in a dose-dependent manner.
CONCLUSIONS: Our findings showed that doses lower than 2 mg/L have repairable effects on gill structure, but the concentration of 2 mg/L cadmium leaves irreparable and destructive effects on the gill tissue.
OBJECTIVE: This study were done to determine the toxicity of cadmium to Litopenaeus vannamei and to evaluate the histological changes in gill tissues after exposure to sublethal concentrations of cadmium at different salinities.
METHODS: For this reason, toxicity test was done to determine the lethal concentration (LC50) of cadmium for whiteleg shrimp. According to the calculated LC50 amount, sublethal doses of cadmium were used to determine its histological effects in different salinity during 2 weeks exposing period.
RESULTS: LC50 of cadmium for 96 h for whiteleg shrimp was 6.56 mg/L. Histological alterations in the gill were observed in L. vannamei after 14 days exposure to different concentrations of cadmium and salinity. Histopathological index was increased in a dose-dependent manner.
CONCLUSIONS: Our findings showed that doses lower than 2 mg/L have repairable effects on gill structure, but the concentration of 2 mg/L cadmium leaves irreparable and destructive effects on the gill tissue.
摘要：
背景：重金属是海洋沿海生态系统中最重要的环境污染物之一。镉是一种重金属，通过工业废物和石油生产活动进入海洋环境。
目的：这项研究是为了确定镉对凡纳滨对虾的毒性，并评估在不同盐度下暴露于亚致死浓度的镉后，g组织的组织学变化。
方法：因此，进行了毒性试验，以确定镉对白对虾的致死浓度（LC50）。根据计算的LC50金额，亚致死剂量的镉用于确定其在2周暴露期间在不同盐度下的组织学作用。
结果：对白虾进行96小时镉的LC50为6.56mg/L。在暴露于不同浓度的镉和盐度14天后，在南美白对虾中观察到g的组织学变化。组织病理学指数以剂量依赖性方式增加。
结论：我们的发现表明，低于2mg/L的剂量对g结构具有可修复的作用，但是浓度为2mg/L的镉对g组织的叶片具有不可修复的破坏作用。
目的：这项研究是为了确定镉对凡纳滨对虾的毒性，并评估在不同盐度下暴露于亚致死浓度的镉后，g组织的组织学变化。
方法：因此，进行了毒性试验，以确定镉对白对虾的致死浓度（LC50）。根据计算的LC50金额，亚致死剂量的镉用于确定其在2周暴露期间在不同盐度下的组织学作用。
结果：对白虾进行96小时镉的LC50为6.56mg/L。在暴露于不同浓度的镉和盐度14天后，在南美白对虾中观察到g的组织学变化。组织病理学指数以剂量依赖性方式增加。
结论：我们的发现表明，低于2mg/L的剂量对g结构具有可修复的作用，但是浓度为2mg/L的镉对g组织的叶片具有不可修复的破坏作用。
