内源性内皮对炎症介质的高反应性驱动克拉克森疾病模型中的急性发作.
关键词: Endothelial cells Vascular biology
Mesh : Animals Nitric Oxide Synthase Type III / metabolism Mice Capillary Leak Syndrome / metabolism pathology Humans Disease Models, Animal Vascular Endothelial Growth Factor A / metabolism genetics Endothelial Cells / metabolism pathology Endothelium, Vascular / metabolism pathology physiopathology Histamine / metabolism Inflammation Mediators / metabolism NG-Nitroarginine Methyl Ester / pharmacology Protein Phosphatase 2 / metabolism genetics Male
来 源:
DOI:10.1172/JCI169137
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