Abstract：
Hypertension, a prevalent chronic ailment, has the potential to impair kidney function, and thereby resulting in hypertensive nephropathy. The escalating incidence of hypertensive nephropathy attributed to the aging population in urban areas, has emerged as a prominent cause of end-stage renal disease. Nevertheless, the intricate pathogenesis of hypertensive nephropathy poses considerable obstacles in terms of precise clinical diagnosis and treatment. This paper aims to consolidate the research findings on the pathogenesis of hypertensive nephropathy by focusing on the perspective of molecular biology.
摘要：
高血压,一种普遍的慢性疾病，有可能损害肾功能，从而导致高血压肾病。高血压肾病的发病率上升归因于城市地区人口老龄化，已成为终末期肾病的主要原因。然而，高血压肾病复杂的发病机制在临床诊断和治疗方面存在相当大的障碍。本文旨在从分子生物学的角度来巩固高血压肾病发病机制的研究成果。
