Abstract：
Becker\'s nevus (BN) manifests as a hyperpigmented, sometimes hypertrichotic plaque/patch over the chest and shoulder, and it is in the category of benign cutaneous hamartomas. BN has elongation and fusion of the rete ridge, keratotic plugging, sebaceous hyperplasia, smooth muscle hyperplasia, and hyperpigmentation of the basal/suprabasal layer histologically. This article highlights all issues involved in pathogenesis and treatment options of BN. According to current research, postzygotic ACTB mutations induce BN and Becker\'s nevus syndrome (BNS). Although several therapy strategies were utilized to treat the pigmentary and hypertrichotic aspects of BN, no definitive standard treatment was identified to far, and further research is needed to better educate BN care.
摘要：
贝克尔的痣（BN）表现为色素沉着过度，有时在胸部和肩膀上出现多毛斑块/斑块，属于良性皮肤错构瘤。BN具有网状脊的伸长和融合，角化堵塞，皮脂腺增生,平滑肌增生,和组织学上基底/上层的色素沉着过度。本文重点介绍了与BN的发病机制和治疗方案有关的所有问题。根据目前的研究,合子后ACTB突变可诱导BN和Becker痣综合征（BNS）。尽管使用了几种治疗策略来治疗BN的色素性和多毛性方面，目前还没有确定明确的标准治疗方法，需要进一步的研究来更好地教育BN护理。
