{Reference Type}: Journal Article
{Title}: Spastic Equinovarus Foot Deformity.
{Author}: Martin KD;Jastifer J;Scott D;Grzeskiewicz E;
{Journal}: J Am Acad Orthop Surg
{Volume}: 32
{Issue}: 16
{Year}: 2024 Aug 15
{Factor}: 4
{DOI}: 10.5435/JAAOS-D-23-01007
{Abstract}: Acute brain injuries are caused by a variety of etiologies, each potentially disrupting neurological function. The neurologic impairments are on a spectrum of severity often creating functional barriers to completing activities of daily living. Initial treatment starts immediately upon diagnosis and requires a multimodal approach working to prevent systemic changes. Therapy, bracing treatment, injections, and pharmacologic treatments are the mainstay of early intervention. Worsening upper motor neurological impairment associated with involuntary muscle hyperactivity can lead to a spastic equinovarus foot deformity. Spastic equinovarus foot deformities secondary to anoxic brain injuries or traumatic brain injury pose a challenging situation for orthopaedic surgeons because of associated cognitive impairment, spastic tone, and extensive soft-tissue contractures prohibiting bracing treatment. Tendon releases and transfers in combination with functional bracing treatment are initially attempted, and selective fusions are performed for severe cases. Surgical indications are primarily focused on obtaining a balanced, braceable, functional lower extremity with a plantigrade foot.