{Reference Type}: Case Reports
{Title}: Type I CD36 deficiency associated with metabolic syndrome and vasospastic angina: a case report.
{Author}: Kamiya M;Nakagomi A;Tokita Y;Yasutake M;Kusama Y;Takayama M;Takano T;
{Journal}: J Cardiol
{Volume}: 48
{Issue}: 1
{Year}: Jul 2006
{Factor}: 2.974
{Abstract}: A 54-year-old man was admitted to our hospital for evaluation of chest pain occurring at rest in the morning. ST segment depression was observed during a treadmill exercise test. Coronary angiography identified spontaneous spasm of the proximal right coronary artery, and right coronary obstruction was improved from 90% to about 50% stenosis after intracoronary administration of nitroglycerin. Myocardial iodine-123 beta-methyl-p-iodophenyl-pentadecanoic acid uptake was absent, but thallium-201 uptake during single photon emission computed tomography was normal, and neither platelet nor monocyte expression of the CD36 molecule was observed, indicating type I CD36 deficiency. High blood pressure, elevated plasma triglyceride and fasting plasma glucose levels, and low high-density lipoprotein values suggested metabolic syndrome. The final diagnosis was type I CD 36 deficiency associated with metabolic syndrome and vasospastic angina.