Abstract：
Preterm infants can face lasting neurodevelopmental challenges due to hypoxia-induced injury of the cerebral white matter. In this issue of Neuron, Ren et al.1 identify microvascular pericytes as unexpected targets for growth hormone signaling, which enhances angiogenesis and remyelination after hypoxic injury in the developing mouse brain.
摘要：
由于缺氧引起的脑白质损伤，早产儿可能面临持久的神经发育挑战。在本期的神经元中,Ren等人将微血管周细胞识别为生长激素信号的意外目标，在发育中的小鼠脑缺氧损伤后增强血管生成和髓鞘再生。
