PDF(Pubmed)
Abstract：
There are different theories cited as the cause of Motion Sickness. Asymmetrical otolithic organ function is one of the etiology theories of motion sickness. In our study, the aim was to reveal whether there is a cause originating from the sacculocolic reflex pathway in the physiopathology of seasickness. The patient group included 15 man sailors with diagnosis of seasickness and the control group included 15 man sailors without seasickness in our study. Cervical evoked myogenic vestibular potential (cVEMP) findings were compared between the groups. In the right and left ears of sailors with seasickness, the mean latencies of the p13 and n23 waves at 100, 95, and 90 dB normal hearing level (nHL) levels were found significantly shorter than in the control group (p < 0.05, for all). In the seasickness group, cVEMP interaural wave amplitude asymmetry ratio at 100 nHL level were found significantly higher than the control group (p = 0.001). The findings of our study indicated that there may be reasons arising rom the sacculocolic reflex pathway in the pathophysiology of seasickness.
摘要：
有不同的理论被引用为运动病的原因。不对称耳石器官功能是晕动病的病因学理论之一。在我们的研究中,目的是揭示在晕船的病理生理学中是否存在起源于囊性反射途径的原因。在我们的研究中，患者组包括15名诊断为晕船的男子水手，对照组包括15名没有晕船的男子水手。比较两组之间的颈诱发肌源性前庭电位（cVEMP）发现。在水手们晕船的左右耳朵里，发现在100、95和90dB正常听力水平（nHL）水平下，p13和n23波的平均潜伏期明显短于对照组（全部p<0.05）。在晕船组，发现100nHL水平的cVEMP耳间波振幅不对称率明显高于对照组（p=0.001）。我们的研究结果表明，在晕船的病理生理学中，可能存在从糖反射途径引起的原因。
