PDF(Pubmed)
Abstract：
Coronavirus disease 2019 (COVID-19) creates acute and long-lasting infection which results in respiratory, cardiovascular, and neuropsychiatric problems. Etiology of neuropsychiatric manifestations can be associated with immune system response, inflammatory cytokines, and also the stressors which are experienced by patients as feeling the risk of being infected by the virus, economic problems, and social distancing. We aimed to present a case of a 53-year-old patient whose suicide note was found and was admitted with depressive and catatonic symptoms 8 weeks after the recovery from COVID-19. Catatonia was suspected, and he was given lorazepam 1 mg. Shortly thereafter, he was entirely alert, cooperative, and oriented. As an advantage of this case, the patient in our report had not used medications for COVID-19 and so we could exclude the effect of medications to the pathophysiology of post- coronavirus disease symptoms. A wide spectrum of neuropsychiatric manifestations was observed in terms of diagnosis after COVID-19. Catatonia can break out in the post-infectious period as well as in the para-infectious period. There are limitations to figure out the direct invasion of coronavirus and the effect of the systemic inflammation to the central nervous system. Nevertheless, it should be considered that catatonia may be one of the clinical results of COVID-19.
摘要：
2019年冠状病毒病（COVID-19）造成急性和持久的感染，导致呼吸道感染，心血管,和神经精神问题。神经精神表现的病因可能与免疫系统反应有关，炎性细胞因子，以及患者感受到被病毒感染的风险的压力源，经济问题,和社交距离。我们的目的是介绍一例53岁的患者，他的遗书被发现，并在从COVID-19中恢复8周后因抑郁和紧张性症状入院。卡塔托尼亚被怀疑，他服用了1毫克的劳拉西泮。此后不久,他完全警觉,合作社,和导向。作为这种情况的优势，我们报告中的患者没有使用治疗COVID-19的药物,因此我们可以排除药物对冠状病毒疾病症状后病理生理学的影响.在COVID-19后的诊断方面观察到了广泛的神经精神表现。紧张症可以在感染后时期以及在感染前时期爆发。了解冠状病毒的直接侵袭和全身性炎症对中枢神经系统的影响存在局限性。然而，应考虑卡通症可能是COVID-19的临床结果之一。
