Abstract：
This is the case of a 26-year-old male who developed Anton Babinski syndrome (ABS), quadriplegia, and delayed post-hypoxic leukoencephalopathy (DPHL) after an opioid overdose. He exhibited cortical blindness, visual anosognosia, and confabulation upon awakening. Several days later, he experienced acute psychosis and agitation. T2-FSE MRI revealed extensive supratentorial leukoencephalopathy involving both cerebral hemispheres, extending to the posterior corpus callosum due to cerebral anoxia. This case report will discuss different types of encephalopathy from opioid abuse, ABS, visual anosognosia, and confabulation\'s pathogenic mechanisms. It underscores the necessity of researching substance-induced neuropsychiatric disorders and their pathogenic mechanisms for effective treatments.
摘要：
这是一个26岁的男性，他患上了安东·巴宾斯基综合征（ABS），四肢瘫痪,和阿片类药物过量后迟发性缺氧后白质脑病（DPHL）。他表现出皮质盲，视觉失认症,醒来后的虚构。几天后,他经历了急性精神病和躁动。T2-FSEMRI显示广泛的幕上白质脑病累及两个大脑半球，由于脑缺氧而延伸到后call体。本病例报告将讨论阿片类药物滥用引起的不同类型的脑病，ABS,视觉失认症,和虚构的致病机制。它强调了研究物质引起的神经精神疾病及其有效治疗的致病机制的必要性。
