Abstract：
Mycoplasma hyopneumoniae is the pathogen causing swine mycoplasmal pneumonia. The lack of well-established animal models of M. hyopneumoniae infection has delayed the progress of M. hyopneumoniae-related anti-infection immunity studies. This paper reviews the inflammatory response, the recognition of M. hyopneumoniae by the innate immune system, and the role of innate immune cells, complement system, antimicrobial peptides, autophagy, and apoptosis in M. hyopneumoniae infection. The aim was to elucidate the important roles played by the components of the innate immune system in the control of M. hyopneumoniae infection, and prospect key research directions of innate immune response of M. hyopneumoniae infection in the future.
摘要：
猪肺炎支原体是引起猪支原体肺炎的病原体。猪肺炎支原体感染的动物模型的缺乏已经推迟了猪肺炎支原体相关抗感染免疫研究的进展。本文就炎症反应,先天免疫系统对猪肺炎支原体的识别，以及先天免疫细胞的作用，互补系统，抗菌肽，自噬,猪肺炎支原体感染和细胞凋亡。目的是阐明先天免疫系统的组成部分在控制猪肺炎支原体感染中起的重要作用。并对猪肺炎支原体感染天然免疫应答的未来研究方向进行了展望。
