Abstract：
UNASSIGNED: Atrioventricular (AV) reentrant tachycardia is a common type of supraventricular tachycardia (SVT) that occurs in the fetus and neonate. Although many tachycardias resolve within several weeks of birth or respond to medical management, disruptions in the cardiac annulus fibrosus and development of additional accessory pathways may lead to refractory dysrhythmia resulting in fetal hydrops and ultimately, fetal death.
UNASSIGNED: While accessory pathways have been well documented anatomically in adult and childhood tachyarrhythmias, there are no reports of the histology of these pathways in human fetuses with SVT.
UNASSIGNED: This is a small case series of 2 fetuses with a history of SVT that resulted in fetal hydrops.
UNASSIGNED: In both cases, examination of the cardiac conduction system was unremarkable and examination of the atrioventricular junction revealed a focally thinned and/or discontinuous annulus fibrosus with documented direct continuity between the atrial and ventricular myocardium in 1 case.
UNASSIGNED: This case series demonstrates that thinning or absence of the annulus fibrosus is a feature seen in fetal SVT, and the development of subsequent aberrant AV connections due to defective formation of the annulus fibrosus suggests a possible cause for these arrhythmias.
UNASSIGNED: While accessory pathways have been well documented anatomically in adult and childhood tachyarrhythmias, there are no reports of the histology of these pathways in human fetuses with SVT.
UNASSIGNED: This is a small case series of 2 fetuses with a history of SVT that resulted in fetal hydrops.
UNASSIGNED: In both cases, examination of the cardiac conduction system was unremarkable and examination of the atrioventricular junction revealed a focally thinned and/or discontinuous annulus fibrosus with documented direct continuity between the atrial and ventricular myocardium in 1 case.
UNASSIGNED: This case series demonstrates that thinning or absence of the annulus fibrosus is a feature seen in fetal SVT, and the development of subsequent aberrant AV connections due to defective formation of the annulus fibrosus suggests a possible cause for these arrhythmias.
摘要：
■房室折返性心动过速是胎儿和新生儿常见的室上性心动过速（SVT）。尽管许多心动过速在出生后几周内消退或对医疗管理有反应，心脏纤维环的破坏和其他辅助途径的发展可能导致难治性心律失常，导致胎儿水肿，最终，死胎.
■虽然辅助通路在成人和儿童快速性心律失常的解剖学上有很好的记载，在患有SVT的人类胎儿中，没有关于这些途径的组织学报道。
■这是一个由2个有SVT病史的胎儿导致胎儿水肿的小病例系列。
■在这两种情况下，在1例中,心脏传导系统的检查没有异常,房室交界处的检查显示纤维环出现局灶性变薄和/或不连续,并记录了心房和心室心肌之间的直接连续性.
■本系列病例显示，纤维环变薄或缺失是胎儿室上性心动过速的特征，由于纤维环的缺陷形成，随后的异常AV连接的发展表明了这些心律失常的可能原因。
■虽然辅助通路在成人和儿童快速性心律失常的解剖学上有很好的记载，在患有SVT的人类胎儿中，没有关于这些途径的组织学报道。
■这是一个由2个有SVT病史的胎儿导致胎儿水肿的小病例系列。
■在这两种情况下，在1例中,心脏传导系统的检查没有异常,房室交界处的检查显示纤维环出现局灶性变薄和/或不连续,并记录了心房和心室心肌之间的直接连续性.
■本系列病例显示，纤维环变薄或缺失是胎儿室上性心动过速的特征，由于纤维环的缺陷形成，随后的异常AV连接的发展表明了这些心律失常的可能原因。
