Abstract：
UNASSIGNED: There is an urgent clinical need to provide a theoretical basis for silver needle thermal therapy to Myofacial pain syndrome (MPS).
UNASSIGNED: This study was conducted to explore the effect of silver needle thermal therapy on myofascial pain syndrome in rats.
UNASSIGNED: MPS rat models were duplicated, and the rats were subsequently divided into model and treatment groups. A normal control group was synchronously set up. No treatment was given to the model group, whereas silver needle thermal therapy was administered to the treatment group. The thermal and mechanical pain threshold, the morphological structure as well as the expression of 5-HT3 receptors in the spinal cord were observed.
UNASSIGNED: Rats from the treatment group presented with a significantly higher pain threshold compared to the untreated model group.
The myofascial arrangement of the affected part of the model group was disordered, and some muscle fibers were atrophied and deformed. Meanwhile, the myofascial arrangement of the treatment group became more regular than that of the model group. The expression levels of 5-HT3 receptor in the spinal cord of the untreated model group were significantly increased, while being markedly decreased in the treatment group.
UNASSIGNED: Silver needle thermal therapy can augment the pain threshold of rats with MPS, repair the damaged myofascial membrane in the rats, and further reduce the expression of 5-HT3 receptors in the spinal cord of the MPS rats.
UNASSIGNED: This study was conducted to explore the effect of silver needle thermal therapy on myofascial pain syndrome in rats.
UNASSIGNED: MPS rat models were duplicated, and the rats were subsequently divided into model and treatment groups. A normal control group was synchronously set up. No treatment was given to the model group, whereas silver needle thermal therapy was administered to the treatment group. The thermal and mechanical pain threshold, the morphological structure as well as the expression of 5-HT3 receptors in the spinal cord were observed.
UNASSIGNED: Rats from the treatment group presented with a significantly higher pain threshold compared to the untreated model group.
The myofascial arrangement of the affected part of the model group was disordered, and some muscle fibers were atrophied and deformed. Meanwhile, the myofascial arrangement of the treatment group became more regular than that of the model group. The expression levels of 5-HT3 receptor in the spinal cord of the untreated model group were significantly increased, while being markedly decreased in the treatment group.
UNASSIGNED: Silver needle thermal therapy can augment the pain threshold of rats with MPS, repair the damaged myofascial membrane in the rats, and further reduce the expression of 5-HT3 receptors in the spinal cord of the MPS rats.
摘要：
UNASSIGNED：临床上迫切需要为银质针热疗法治疗肌面部疼痛综合征（MPS）提供理论依据。
UNASSIGNED：本研究旨在探讨银针热疗法对大鼠肌筋膜疼痛综合征的影响。
未经证实：复制MPS大鼠模型，随后将大鼠分为模型组和治疗组。同步设置正常对照组。模型组不给予治疗,而银质针热疗则给予治疗组。热和机械痛阈值，观察脊髓形态结构及5-HT3受体的表达。
UNASSIGNED:与未处理的模型组相比,来自处理组的大鼠呈现显著更高的疼痛阈值。
模型组患处肌筋膜排列紊乱，一些肌肉纤维萎缩变形.同时,治疗组的肌筋膜排列比模型组更加规则。未治疗模型组大鼠脊髓5-HT3受体表达水平显著升高,而治疗组明显下降。
UNASSIGNED：银针热疗可增加MPS大鼠的痛阈，修复大鼠受损的肌筋膜，并进一步降低MPS大鼠脊髓中5-HT3受体的表达。
UNASSIGNED：本研究旨在探讨银针热疗法对大鼠肌筋膜疼痛综合征的影响。
未经证实：复制MPS大鼠模型，随后将大鼠分为模型组和治疗组。同步设置正常对照组。模型组不给予治疗,而银质针热疗则给予治疗组。热和机械痛阈值，观察脊髓形态结构及5-HT3受体的表达。
UNASSIGNED:与未处理的模型组相比,来自处理组的大鼠呈现显著更高的疼痛阈值。
模型组患处肌筋膜排列紊乱，一些肌肉纤维萎缩变形.同时,治疗组的肌筋膜排列比模型组更加规则。未治疗模型组大鼠脊髓5-HT3受体表达水平显著升高,而治疗组明显下降。
UNASSIGNED：银针热疗可增加MPS大鼠的痛阈，修复大鼠受损的肌筋膜，并进一步降低MPS大鼠脊髓中5-HT3受体的表达。
