SEMA3C 通过神经丛蛋白 B1 反式激活多种受体酪氨酸激酶来驱动癌症生长。
关键词: Plexin B1 Semaphorin 3C apoptosis prostate cancer receptor tyrosine kinase
Mesh : Animals Cell Proliferation Humans Male Mice Nerve Tissue Proteins / metabolism Prostatic Neoplasms, Castration-Resistant / metabolism pathology Receptor Protein-Tyrosine Kinases / metabolism Receptors, Cell Surface / metabolism Semaphorins / antagonists & inhibitors metabolism Signal Transduction Xenograft Model Antitumor Assays
来 源:
DOI:10.15252/emmm.201707689
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